Chinese Medicated Diet

The Chinese have a traditional belief in the medicinal value of food, as they believe that food and medicine share the same origin. This view could be considered a forerunner of nutritional science in China.
Chinese medicated diet is not a simple combination of food and Chinese herb, but a special highly finished diet made from Chinese herbs, food and condiments under the theoretical guidance of diet preparation based on differentiation of symptoms and signs of traditional Chinese medicine.
Chinese medicated diet has a long history. The ancient legend "Shennong Tastes a Hundred Grasses "shows that early in remote antiquity the Chinese nation began to explore the function of food and medicaments, hence the saying "Traditional Chinese medicine and diet both originate from the practice and experience in daily life."
In Shennong's Herbal Classic, which was published approximately in about the Qin and Han Periods and is the extant earliest monograph on materia medica, many sorts of medicaments which are both drugs and food were recorded, such as Chinese-date (Fructus Ziziphi Jujubae),sesame seed (Semen Sesami), Chinese yam (Rhizoma Dioscoreae), grape (Vitis), walnut kernel (Semen Fuglandis), lily bulb (Bulbus Lilii) , fresh ginger (Rhizoma Zingiberis Recens), Job's-tears seed (Semen Coicis), etc. in the East Han dynasty, some noted medicated diet recipes were recorded, such as Soup of Chinese Angelica root, Fresh ginger and Mutton (Danggui Shengjiang Yangrou Tang ), Decoction of Pig-skin (Zhufu Tang), etc., all of which now still have important values. Sun simiao, a well-known doctor in the Tang Dynasty, listed and discussed such questions as dietetic treatment, dietetic treatment for senile health care and health preservation.
According to history books, up to the period of the Sui and Tang Dynasties about more than sixty kinds of books on dietetic treatment had been published. But unfortunately most of them are lost. Of all the prescriptions recorded in it, 70% are about medicated diet. It is emphasized in this book that "dietetic therapy should go first for any senile diseases, and then followed by medicine if they are not cured." Hu Sihui, a royal doctor in the Yuan Dynasty, oceans of medicated diet prescriptions and dietetic drugs were recorded; in addition, some questions, such as diet contraindication in pregnancy, diet contraindication for wet nurse, contraindication for drinking, etc. were also discussed in this book. In the Ming Dynasty, Li Shizhen collected and recorded many medicated diet prescriptions, dozens of which were about medicated gruel alone, another dozens touched on nothing other than medicated wine. Monographs on medicated diet treatment in the Qing Dynasty varied in characteristics. Over 300 species belonging to 7 phyla of medicated food and drink were introduced.
Medicated diet can be used either to treat diseases or for healthy people to build up their health and prevent diseases. This is one of the characteristics in which medicated diet is different from treatment by medicine. Although medicated diet is something mild, it has a notable effect on the prevention and cure of diseases, health building -up and health preserving. Here are some of the achievements in scientific research of Shandong Traditional Chinese Medicine College:
Eight-Ingredient Food: It is prepared according to the experience in ancient dietetic treatment and health care of imperial court in the Qing Dynasty from eight dietetic Chinese drugs including Chinese yam (Rhizoma Dioscoreae), lotus seed (semen Nelumbinis), and hawthorn fruit (Fructus Crataegi). 97% of the children who took it for 30 days have whetted their appetite, and their growth has improved too.
Nourishing Extract of laiyang Pear and mushroom: It is made from the juice of Laiyang Pear (Malum Piri) and extract of mushrooms ( Lentinus Edodes) and tremella (Tremella). If the middle-aged and senile patients suffering from chronic diseases take it, not only can the symptoms of their illness be alleviated, but their blood-fat can be brought down too when they are suffering from hyperlipemia, and their immunology function can be improved.
It has not only the efficiency of medicine but also the delicacy of food, and can be used to prevent and cure diseases, build up one's health and prolong one's life.

How to Prevent Membranous Nephropathy

In order to prevent Membranous Nephropathy, patients should pay attention to the following points in the daily life:
1. In the daily life, patients should have a good rest, avoid tiredness, and prevent infections. Taking low protein diet mainly, and supplementing vitamins is also very important. In addition, patients should also avoid taking medicines which will damage the kidneys.
2. In order to direct the conduct of the treatment, patients should return to visit the doctor every one or two weeks during the period of medicine treatment, so as to observe whether the liver and kidney functions are normal by a urinary routine test; and the growth and development of children with Membranous Nephropathy should be especially paid attention to.
3. After the active lesions have been controlled, and the course of treatment has been completed, patients should do a biopsy once again, so as to observe the pathological changes of the kidney tissues, and estimate whether there is chronic lesion, thus adopting measures timely.
4. Patients should pay attention to protect the remaining kidney functions, correct kinds of factors which decrease the renal blood flow(such as hypoproteinemia, dehydration, hypotension,etc.) and prevent infections. All these measures mentioned above are important links which can not be ignored in preventing Membranous Nephropathy.
Complications of Membranous Nephropathy which can affect the curative effect and the long-term prognosis of patients should be treated actively:
1. Infections
By the treatment of hormone, patients are easy to suffer from infections. Once infections occur, antibiotic which are sensitive to pathogenic bacteria, effective and has no renal toxicity should be adopted for the treatment, and the definite focal infection should be removed as soon as possible.
2. Thrombus and embolism complications
It is generally acknowledged that when the concentration of plasma albumin is below 20 g/L, hypercoagulable state may occur. Then the preventive anticoagulation should be adopted immediately.
3. Acute Kidney Failure
If Nephrotic Syndrome combined with acute Kidney Failure can not be managed properly, the patient's life will be in danger. However, if the situation can be managed properly, most of the patients can recover.


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What causes Minimal Change Nephropathy?

  Minimal Change Nephropathy is also known as Lipoid Nephrosis. It is a disease of the kidney that causes Nephrotic Syndrome and usually affects children (peak incidence at 2–3 years of age). It is a type of kidney disease where the kidneys leak protein.
Minimal Change Disease is most common in very young children but also can occur in older children and adults. It is by far the most common cause of Nephrotic Syndrome (NS) in children under 10 years old, accounting for the majority (about 90%) of these diagnoses. Among teenagers who develop NS, it is caused by minimal change disease about half the time. It can also occur in adults but accounts for less than 20% of adults diagnosed with NS. Among children less than 10 years old, boys seem to be more likely to develop Minimal Change Disease than girls.
People with 1 or more autoimmune disorders are at increased risk of developing Minimal Change Disease. Having Minimal Change Disease also increases the chances of developing other autoimmune disorders. So from the above-mentioned information we can conclude that the Minimal Change Nephropathy does have an obvious negative impact on little children. So what causes Minimal Change Nephropathy?
It is called minimal change because the kidney looks normal under the microscope. When blood flow through the kidneys, it is filtered to produce urine. The membrane that performs the filtration process is a very delicate structure. A slight abnormality in the structure can cause protein molecules that are normally too large to get through to appear in the urine. Although the membrane looks normal under the microscope, there may be a slight change in the electrical charge that causes this protein leakage. Many patients with minimal change nephropathy have mild allergic conditions such as eczema, but it has not been possible to show that minimal change itself is an allergy.
The pathogenesis of minimal change nephropathy is still unknown, and this disease is characterized by loss of negative charge on glomerular capillary wall, and it also can be developed during kidney transplant. So it supports the idea that the humoral factor which consumes negative charge of glomerulus may exist in the circulating blood, the humoral factor effects electrostatic barrier of glomerulus and then leads to selective proteinuria.
In general, the causes of Minimal Change Nephropathy are complicated. And patients shall pay more attention on nursing, although prognosis of Minimal Change Nephropathy is favorable, the poor therapy or irresponsible care will affect children’ development and aggravate the disease.

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The Causes and Proper Diet of Patients with Diabetic Nephropathy

  “What should we pay attention to the diet of Diabetic Nephropathy and how it causes?” etc., which are the concerned problems of most of patients, here we will give you a brief introduction that is “What should we pay attention to the diet of Diabetic Nephropathy and how it causes?”

What should we pay attention to the diet of Diabetic Nephropathy?

I Diabetic Nephropathy can cause dysbolism of fat cholesterol, and promote incrassation, sclerosis, degeneration of glomeruli and membrane within kidney blood capillary, making them lose their normal function. The keys to control blood sugar are as following: first, restrain the ingestion of calories strictly, second, insist taking glucose-lowering medicines, in order to avoid excite emotion, infection and other various factors which cause fluctuations of blood sugar.

II the ingestion of salt should be restrained. To protect kidney and reduce its workload, dishes of patients with Diabetes should be as insipid as possible, and the number of salt intake should be less than seven grams, and even the quantity of ingestion of water should be restrained, when kidney disease is serious.

III Restrain the ingestion of potassium and protein appropriately. Because Acidosis and Hyperkalemia are very easy to occur for DN patients, once appearing, the disorder of heart rhythm and hepatic coma will be caused to happen.

IV The ingestion of vitamin and microelement should be sufficient, in particular to vitamin B, vitamin C and Zinc, Calcium, Iron etc., which can protect kidneys.

The recommendation of Diabetic Nephropathy diet:

I Soybean and its products: Besides full of protein, mineral salt, vitamin in this kind of food, there are more unsaturated fatty acid in soya-bean oil, which can not only reduce blood cholesterol, but also decrease blood triglyceride, and the contained sitosterol also has lipid-lowering action.

II Coarse mixed sugar: such as hullessoat flour, soba noodles, hot oatmeal, cornmeal contain multiple microelements, vitamin B and edible fibre. The demonstration is that they have the action of delaying the increase of blood sugar.

III Patients with Diabetes should have fruit as few as possible or not eat it at all. Because fruits contain more carbohydrate, which mainly includes glucose, sugar and starch. The digestive and absorptive speed are fast after meal, leading to the increase of blood sugar rapidly, which is harmful to patients with Diabetes.

IV It is necessary for patients with Diabetes to restrain the purity of cholesterol on your diet. The reason is that when the state of patients with Diabetes is controlled without satisfaction, serum cholesterol rises easily, causing vascular complications of Diabetes and coronary disease etc.

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Diabetic Nephropathy and Its Symptoms

Diabetic Nephropathy (DN), a clinically common and frequently-occurring diabetic complication, is one of the most severe complications of Diabetes. It is a major microvascular complication of Diabetes and mainly refers to diabetic glomerular sclerosis, a glomerular lesion with vascular damage as its principal feature. Generally, there is no symptom at the early stage, with the blood pressure normal or higher than normal. The incidence will increase with the prolongation course of Diabetes. Renal changes at the early stage of Diabetes include kidney enlargement，rise in Glomerular filtration rate (GFR) and a high GFR state and intermittent proteinuia or micro-albuminuria will gradually occur. Along with the prolongation of the course of DN, there will be continuous proteinuria, edema, high blood pressure and decrease of GFR, followed by renal inadequacy and uremia, which is the main cause of death in Diabetes. Usually Ganoderma Sinense is used to prevent or treat DN.

Symptoms

I Proteinuria. There is no clinical proteinuria at the early stage of DN, and only with radio-immunity method can micro-albunminuria be detected. Clinically, the only manifestation of early-stage DN is proteinuria, which gradually develops from intermittent to continuous.

II Edema. Usually there is no clinical edema at the early stage of DN. Mild edema may occur in a small number of patients before their plasma protein is lowered. Massive proteinuria, with low plasma protein and aggravated edema, is usually a manifestation that the disease has reached an advanced stage.

III Hypertension. Compared with healthy people, incidence rate of hypertension in patients with Type-1 Diabetes but without Nephropathy is not increased, while in patients with Type-2 Diabetes, more are accompanied by hypertension. But when proteinuria occurs, hypertension rate will also increase. Patients with Nephropathy Syndrome will have hypertension, most of which are moderate and only a small part are severe.

IV Renal failure. Progression speed of DN is greatly differentiated. Slight proteinuria may last for many years for some patients with normal renal function, while some patients who have little urine protein may develop rapidly into Nephropathy Syndrome with renal function deteriorating gradually, and Uremia occurs eventually.

V Anemia. Slight anemia may occur in patients with obvious Azotemia.

VI Complication manifestations of other organs. These complications include cardiovascular lesions such as Cardiac Failure and Myocardial Infarction, neural lesions such as peripheral neuropathy, and neurogenic bladder caused by involvement of autonomic nerves. Retinopathy will almost certainly occur when DN becomes severe. However, severe retinopathy is not necessarily accompanied with obvious renal lesions. Retinopathy deterioration is usually quickened with the progression of DN.

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Manifestations of Lupus Nephritis

  Systemic Lupus Erythematosus(SLE) is a kind of autoimmune disease, which is caused by the generation of a series of autoantibodies duing to the immune disorders. The incidence of this disease is about 0.1%, and the proportion of males and females is 1:8. Almost all the organs in the human body are involved by the lesions of SLE, and the most commonly involved is the kidney. By biopsy, it is found that kidney lesions occur in almost all SLE patients. And when this pathological change occurs in SLE patients, it is called Lupus Nephritis. Then, what are the specific manifestations of Lupus Nephritis?
1. Systemic manifestation
· Intermittent fever
· Malar erythema: as its shape is similar to that of a butterfly, it is also called butterfly erythema.
· Painless oral ulcers
· Swelling and pain occur on the joint.
·Epilepsy or mental disorder occurs.
· Encountering cold, the hands and feet will become pale, and purplish red when they are warm, and finally recover to the normal complexion. This manifestation is also called Raynaud's phenomenon.
2. Renal manifestation
In life, patients with Lupus Nephritis usually show one of the following manifestations:
· Isolated hematuria or proteinuria
· Hematuria or proteinuria with edema, mild lumbago or hypertension, which are similar to the manifestations of Nephritis.
· Large quantities of proteinuria, hypoproteinemia and edema, which are similar to the manifestations of Nephrotic Syndrome.
· Hematuria and proteinuria with rapid decline of renal functions, which present as the manifestations of Rapidly Progressive Glomerulonephritis.
· Manifestations which are similar to those of Chronic Kidney Failure.
3. Laboratory abnormalities
· The occurrence of Leukopenia(< 4.0 ×10/L), anemia or thrombocytopenia(< 100 ×10/L)
· The erythrocyte sedimentation rate(ESR) is rapid.
·The level of complement C3 is low.
·The antinuclear antibody and autoantibody are positive.
Sometimes, the manifestations of Lupus Nephritis are not typical. Therefore, when only the kidney show abnormal symptoms, Lupus Nephritis is often misdiagnosed as Nephritis, Nephrotic Syndrome or Chronic Kidney Failure. And as a result, patients will miss the best time for treatment, and Kidney Failure, Heart Failure, Septicemia or other adverse consequences will occur in patients with Lupus Nephritis. Therefore, a early diagnosis and treatment is very necessary.







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What Are the Causes of Purpura Nephritis

  Henoch-Schonlein Purpura is a kind of syndrome characterized by skin purpura, hemorrhagic gastroenteritis, arthritis and kidney damages. The basic lesion is systemic necrotizing vasculitis. With kidney damages, it is called Henoch-Schonlein Purpura Nephritis, and Purpura Nephritis for short.
Purpura Nephritis can occur at any age, but it is more common in children and teenagers with the age between 5~15. And it is rarely seen in adults who are more than 20 years old. This disease usually occurs in cold seasons.
The cause of Purpura Nephritis is not clear yet, but it has relations with infections and allergic reaction:
1. Before the onset of Purpura Nephritis, about 1/3 of the patients suffer from infections, and the most common is the upper respiratory tract infection. Bacteria infection, virus infection, blood flukes infection or other parasitic infections can also be seen in patients.
2. About 1/4 of the patients have a history of drug allergy. And the commonly seen drugs are antibiotics, sulfonamides, isoniazide, salicylic acid, quinine, etc.
3. Food allergy: fishes, shrimps, crabs, eggs, milk, etc.
4. Other allergies caused by pollen, insect bite or cold stimulation.
The pathogenesis of Purpura Nephritis is not clear yet, but it is believed that the abnormal immune response is the main pathogenesis of this disease. The reason is that before the onset of this disease, patients usually have allergies caused by definite foreign substances. When the disease attacks, the level of IgA in the serum increases in most patients; and after the attack, the deposits of immunoglobulin and complements can be seen in glomeruli and skins. Data indicates that IgA plays an important role in the pathogenesis of Purpura Nephritis. As for the exact mechanism of IgA in the onset of this disease is not clear yet, in some people's opinion, it may be antigen, antibody, or non-specific antibody.
In addition, the recent studies show that the onset of Purpura Nephritis have relations with genetic predisposition.


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Can Female Diabetic Nephropathy Patients Be Pregnant?

  This is a huge problem for female patients with Diabetic Nephropathy, that is they want to know whether they can be pregnant or not and what may happened to their bodies and fetuses during gestation period. Here we will analyze the situations for Diabetic Nephropathy patients who want to have a baby:

First of all, female patients with Diabetic Nephropathy under the following situations can not be Pregnant:

I Some Diabetic Nephropathy with symptoms of Nephrotic Syndrome may lead to Intrauterine Growth Retardation and premature delivery because of Hypoalbuminema.

II The incidence of Intrauterine Growth Retardation and premature delivery of moderate renal insufficiency patients are high, and pregnant woman may experience progressive exacerbation.

III For female patients with Diabetic Nephropathy, some problems such as Preeclampsia, premature delivery, fetal anomaly, macrosomia and Fetal Respiratory Distress Syndrome etc. are likely to happen. So they had better not conceive.

For the pregnant patients with Diabetic Nephropathy, the following points should also be paid attention to:

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What Are Main Pathogeny of Diabetic Nephropathy

  Nowadays, people pay more and more attention to health. As one of the most common disease, Diabetic Nephropathy threatens our life at every moment. So it is necessary for us to learn something about Diabetic Nephropathy to prevent us from developing this disease.

The following are main pathogeny of Diabetic Nephropathy:

I High protein diet: High protein diet will aggravate Diabetic Nephropathy. Diabetic Nephropathy patients shall limit the intake of Carbohydrate strictly, and daily meals should be based on food with high protein and fiber. If we do not follow this, too much protidtemns and phosphorus load will aggravate pathological damages of Diabetic Nephropathy.

  II Effect of high blood pressure: many diabetics suffer from hypertension because of lipid metabolic disturbance, atherosclerosis and other reasons. Most of them have microalbuminuria, which is an indicator that kidneys are damaged.

III High blood sugar: long-term and excessive high blood sugar will lead to increase of capillary permeability and exosmosis of plasma protein, and then cause damage of glomerular capillary basement membrane and shrinking of renal tissues.

According to extent of damage, Diabetic Nephropathy can be classified into 5 stages.

Stage I: this stage is characterized by high glomerular filtration rate and large kidneys’ size. Meanwhile, renal blood flow, glomerular capillary perfusion and glomerular internal pressure will also increase.

Stage II: Urinary Albumin Excretion Rate (UAER) is normal in this stage (<20 μg/min or <30 mg/24h). UAER will increase after exercise and drop to normal range after rest.

Stage III: early stage of Diabetic Nephropathy. This stage is characterized by continued higher level of UAER (20～200 μg/min or 30～300 mg/24h), and high GFR maybe one of reasons of sustained microalbuminuria.

Stage IV: this stage is called clinical Diabetic Nephropathy or overt Diabetic Nephropathy, which is characterized by microalbuminuria, high UAER (UAE>200 μg/min) or sustained high urine protein (>0.5 g).

Stage V: stage of renal failure. Various symptoms and complications of renal failure occur, which are main causes of death.

We will know much about our own body conditions if we understand pathogeny of Diabetic Nephropathy. Meanwhile, Patients with Diabetes should also care about their diet and keep doing physical exercise. Some simple and effective methods are necessary to follow to fight against Diabetic Nephropathy.

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What causes Minimal Change Nephropathy?

Minimal Change Nephropathy is also known as Lipoid Nephrosis. It is a disease of the kidney that causes Nephrotic Syndrome and usually affects children (peak incidence at 2–3 years of age). It is a type of kidney disease where the kidneys leak protein.
Minimal Change Disease is most common in very young children but also can occur in older children and adults. It is by far the most common cause of Nephrotic Syndrome (NS) in children under 10 years old, accounting for the majority (about 90%) of these diagnoses. Among teenagers who develop NS, it is caused by minimal change disease about half the time. It can also occur in adults but accounts for less than 20% of adults diagnosed with NS. Among children less than 10 years old, boys seem to be more likely to develop Minimal Change Disease than girls.


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What Is the Cause of Childhood Nephrotic Syndrome?

Childhood Nephrotic Syndrome(Childhood NS) is a common disease with a long course. It is a group of clinical syndrome caused by various causes which will lead to renal fibrosis, and during the course of renal fibrosis, the increase of glomerular capillary permeability leads to the formation of large amounts of proteinuria. The main manifestations of this disease are large amounts of proteinuria, hypoproteinemia, hyperlipoidemia and edema at different degrees. The edema of Childhood NS presents as pitting edema over the legs.

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Nephrotic Syndrome in Children

Childhood Nephrotic Syndrome, also known as pediatric kidney disease, is one of the urinary system diseases and is commonly seen. Childhood Nephrotic Syndrome is common in children whose age is between 3~6 years old. And it is more common in boys than in girls. The pathogenesis of this disease is not clear yet. Childhood Nephrotic Syndrome is easy to relapse and long delay. And the course of this disease is long.
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Nephrotic Syndrome Overview

Nephrotic Syndrome (NS) is a group of clinical syndrome of Glomerular disease caused by various pathogenies and pathologic types. It is a nonspecific disorder in which the kidneys are damaged, causing them to increase glomerular basement membrane permeability, decrease glomerular filtration rate (GFR) and leak large amounts of protein (proteinuria at least 3.5 grams per day) from the blood into the urine. In general, Nephrotic Syndrome is defined by its primary diseases that attack the kidney's filtering system. It is not a kind of simplex disease. ...

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